By Roger S. Kirby MD FRCS(Urol) FEBU
It is a condensed model of the author's winning Atlas of Prostatic Disease. The emphasis is on a succinct description of prostatic disorder in a mostly illustrative structure. The e-book covers the motives, prognosis and staging of prostate melanoma, benign prostatic hyperplasia and prostatitis. The sections on therapy were superior by way of the addition of algorithms to summarize the decision-making approach.
Men's well-being is now an immense public health and wellbeing factor and prostatic illness may have a big influence at the caliber of lifetime of many males. This publication will supply a handy precis for urologists, basic care physicians and nurse practitioners
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It is a condensed model of the author's profitable Atlas of Prostatic affliction. The emphasis is on a succinct description of prostatic sickness in a mostly illustrative layout. The e-book covers the factors, analysis and staging of prostate melanoma, benign prostatic hyperplasia and prostatitis. The sections on remedy were greater via the addition of algorithms to summarize the decision-making method.
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Additional info for An illustrated pocketbook of prostatic diseases
Benign prostatic hyperplasia Histological benign prostatic hyperplasia is classically characterized by a mixed proliferation of both stromal and epithelial elements to form nodules (Figure 30). There are, however, individual variations, with some patients developing a predominantly stromal version of the disease and others showing mainly epithelial overgrowth (Figure 31). The luminal-to-basal cell relationship is retained in epithelial hyperplasia, which is not considered to be a premalignant condition.
One explanation for this may be that peripheral zone prostatic ducts enter the prostatic urethra at a less oblique angle than do central zone ducts. This may render the peripheral zone ducts more susceptible to intraprostatic reflux of urine during micturition (Figure 25). Studies using intravesical instillation of India ink prior to voiding and subsequent prostatectomy have revealed an intraprostatic finding of carbon particles (Figure 26) and have also demonstrated their subsequent ingestion by macrophages23 (Figure 27).
When bladder outlet obstruction is severe and bladder emptying is impaired, diverticular formation may develop, together with the formation of bladder calculi; only rarely these days do bilateral hydronephrosis and renal impairment occur (Figure 48). Figure 47 Collagen deposition has occurred between the smooth muscle cell bundles of the bladder detrusor muscle. This phenomenon is seen as part of the bladder response to the gradual development of obstruction (Masson trichrome) 48 AN ILLUSTRATED POCKETBOOK OF PROSTATIC DISEASES Figure 48 Secondary effects of obstructive benign prostatic hyperplasia include bladder wall hypertrophy with diverticula and bladder stone formation.